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Classical ferroptosis inducers act through convergent mechanisms: erastin inhibits system Xc- and depletes glutathione, whereas RSL3 directly inhibits GPX4, a key enzyme that reduces lipid hydroperoxides (23) (Figure 5)

This underscores the necessity for diversified, disease-specific NAD + -targeting strategies (Liu et al

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While the exact cause of uterine leiomyoma formation is still unknown, epidemiological studies have provided substantial evidence that estrogen and progesterone stimulate the growth of the tumor, possibly as a result of continuous menstrual cycles, since fibroids rarely develop prior to menarche and disappear following menopause [2]