Rat Myogenin, MYOG ELISA Kit, 96T Peptide Synthesis CERKL was initially identified as

CERKL was initially identified as a locus (RP26) associated with an autosomal recessive form of retinitis pigmentosa (arRP) disease

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Constitutive overexpression of human stanniocalcin 2 in mice resulted in pre- and postnatal growth restriction

The protease domain of this protein is thought to be cleaved and secreted into cell media after autocleavage

translocates into the mitochondria where it promotes mtDNA transcription

Rat Myogenin, MYOG ELISA Kit, 96T Peptide Synthesis CERKL was initially identified asActs as a transcriptional activator that promotes transcription of muscle specific target genes and plays a role in muscle differentiation, cell cycle exit and muscle atrophy. Essential for the development of functional embryonic skeletal fiber muscle differentiation. However is dispensable for postnatal skeletal muscle growth; phosphorylation by CAMK2G inhibits its transcriptional activity in respons to muscle activity. Required for the recruitment